[1] GRAHAM WV, HE W, MARCHIANDO AM, et al. Intracellular MLCK1 diversion reverses barrier loss to restore mucosal homeostasis[J]. Nat Med, 2019, 25(4): 690. doi: 10.1038/s41591-019-0393-7
[2] DUAN C, KUANG L, XIANG X, et al. Activated Drp1-mediated mitochondrial ROS influence the gut microbiome and intestinal barrier after hemorrhagic shock[J]. Aging (Albany NY), 2020, 12(2): 1397.
[3] SHEN W, WOLF PG, CARBONERO F, et al. Intestinal and systemic inflammatory responses are positively associated with sulfidogenic bacteria abundance in high-fat-fed male C57BL/6J mice[J]. J Nutr, 2014, 144(8): 1181. doi: 10.3945/jn.114.194332
[4] DEITCH EA, XU D. Role of the gut in the development of injury- and shock induced SIRS and MODS: the gut-lymph hypothesis, a review[J]. Front Biosci, 2006, 11(1): 520. doi: 10.2741/1816
[5] DE MORENO DE LEBLANC A, DEL CARMEN S, ZURITA-TURK M, et al. Importance of IL-10 modulation by probiotic microorganisms in gastrointestinal inflammatory diseases[J]. ISRN Gastroenterol, 2011, 2011(1): 892971.
[6] ZHOU M, XU W, WANG J, et al. Boosting mTOR-dependent autophagy via upstream TLR4-MyD88-MAPK signalling and downstream NF-κB pathway quenches intestinal inflammation and oxidative stress injury[J]. EBioMedicine, 2018, 35: 345. doi: 10.1016/j.ebiom.2018.08.035
[7] 宋琦, 徐志鹏, 王振杰, 等. 醋酸钠林格液复苏失血性休克大鼠对肺炎性介质及其JNK信号通路的影响[J]. 中华急诊医学杂志, 2018, 27(6): 638. doi: 10.3760/cma.j.issn.1671-0282.2018.06.014
[8] YADAV VR, HUSSAIN A, SAHOO K, et al. Remediation of hemorrhagic shock-induced intestinal barrier dysfunction by treatment with diphenyldihaloketones EF24 and CLEFMA[J]. J Pharmacol Exp Ther, 2014, 351(2): 413. doi: 10.1124/jpet.114.217331
[9] WU XJ, LIU HM, SONG XM, et al. Penehyclidine hydrochloride inhibits TLR4 signaling and inflammation, and attenuates blunt chest trauma and hemorrhagic shock-induced acute lung injury in rats[J]. Mol Med Rep, 2018, 17(5): 6327.
[10] YU HP, HWANG TL, HSIEH PW, et al. Role of estrogen receptor-dependent upregulation of P38 MAPK/heme oxygenase 1 in resveratrol-mediated attenuation of intestinal injury after trauma-hemorrhage[J]. Shock, 2011, 35(5): 517. doi: 10.1097/SHK.0b013e318209e931
[11] CHANG SY, SUN RQ, FENG M, et al. BML-111 inhibits the inflammatory response and apoptosis of renal tissue in rats with hemorrhagic shock by inhibiting the MAPK pathway[J]. Eur Rev Med Pharmacol Sci, 2018, 22(11): 3439.
[12] BARRENSCHEE M, LEX D, UHLIG S. Effects of the TLR2 agonists MALP-2 and Pam3Cys in isolated mouse lungs[J]. PLoS One, 2010, 5(11): e13889. doi: 10.1371/journal.pone.0013889
[13] HSU JT, CHEN TH, CHIANG KC, et al. Role of p38 MAPK pathway in 17β-estradiol-mediated attenuation of hemorrhagic shock-induced hepatic injury[J]. J Appl Physiol, 2015, 118(2): 187. doi: 10.1152/japplphysiol.00464.2014
[14] 王振杰, 徐志鹏, 陈硬, 等. 醋酸钠林格液对失血性休克大鼠心肌炎性介质及NF-κB、MAPK信号通路的影响[J]. 蚌埠医学院学报, 2020, 45(3): 281.
[15] 徐志鹏, 陈硬, 宋琦, 等. 醋酸钠林格液联合乌司他丁对失血性休克大鼠肝组织NF-κB p65蛋白表达及其细胞因子的影响[J]. 蚌埠医学院学报, 2018, 43(10): 1334.
[16] 朱然. 应用微透析检测创伤/失血性休克肠道代谢及其参与肠屏障损伤机制的研究[D]. 沈阳: 中国医科大学, 2018.
[17] PATEL R, DUPONT HL. New approaches for bacteriotherapy: prebiotics, new-generation probiotics, and synbiotics[J]. Clin Infect Dis, 2015, 60(Suppl 2): S108.