[1] 陈伟伟, 高润霖, 刘力生, 等.中国心血管病报告2017概要[J].中国循环杂志, 2018, 33(1):1.
[2] PIEPOLI MF, HOES AW, AGEWALL S, et al.2016 European Guidelines on cardiovascular disease prevention in clinical practice[J].Rev Esp Cardiol(Engl Ed), 2016, 69(10):939.
[3] 胡艺川.乙醛脱氢酶2可减少氧化损伤诱导的心肌细胞凋亡[J].基因组学与应用生物学, 2019, 38(3):1417.
[4] CHEN CH, SUN L, MOCHLYROSEN D.Mitochondrial aldehyde dehydrogenase andcardiac diseases[J].Cardiovasc Res, 2010, 88(1):51.
[5] LIU B, WANG J, LI M, et al.Inhibition of ALDH2 by O-GlcNAcylation contributes to the hyperglycemic exacerbation of myocardial ischemia/reperfusion injury[J].Oncotarget, 2017, 8(12):19413. doi: 10.18632/oncotarget.14297
[6] KODA K, SALAZAR-RODRIGUEZ M, CORI F, et al.Aldehyde dehydrogenase activation prevents reperfusion arrhythmias by inhibiting local renin release from cardiac mast cells[J].Circulation, 2010, 122(8):771. doi: 10.1161/CIRCULATIONAHA.110.952481
[7] MA H, GUO R, YU L, et al.Aldehyde dehydrogenase 2(ALDH2) rescues myocardial ischaemia/reperfusion injury:role of autophagy paradox and toxic aldehyde[J].Eur Heart J, 2011, 32(8):1025. doi: 10.1093/eurheartj/ehq253
[8] ALTAF A, QU P, ZHAO Y, et al.NLRP3 inflammasome in peripheral blood monocytes of acute coronary syndrome patients and its relationship with statins[J].Coron Artery Dis, 2015, 26(5):409. doi: 10.1097/MCA.0000000000000255
[9] 王文连, 曹瑞平, 林欣, 等.激动线粒体乙醛脱氢酶2可降低高糖诱导的A549细胞炎性小体生成[J].卫生研究, 2019, 48(3):458.
[10] 陈娟, 林玉壁, 尹更生, 等.血清ACE2水平在冠心病发生发展中的变化[J].中国病理生理杂志, 2017, 33(6):1086. doi: 10.3969/j.issn.1000-4718.2017.06.021
[11] 朱建, 杨燕, 胡司淦, 等.Connexin 43在急性冠脉综合征病人外周血单核细胞中的表达[J].南方医科大学学报, 2019, 39(4):471.
[12] GENSINI GG.A moremeaningful scoring system for determining the severity of coronary of coronary heart disease[J].Am J Cardiol, 1983, 51(3):606.
[13] ROFFI M, PATRONO C, COLLET JP, et al.2015 ESC guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation:Task force for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation of the European Society of Cardiology(ESC)[J].Eur Heart J, 2016, 37(3):267. doi: 10.1093/eurheartj/ehv320
[14] ROFFI M, PATRONO C, COLLET JP, et al.2015 ESC guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation[J].Kardiol Pol, 2015, 73(12):1207.
[15] NARIT M, KITAGAWA K, NAGAI Y, et al.Effects of aldehyde dehydro genase on carotid atherosclerosis[J].U ltrasound Med Biol, 2003, 29(10):1415. doi: 10.1016/S0301-5629(03)01015-9
[16] CHEN CH, BUDAS GR, CHURCHILL EN, et al.Activation of aldehyde dehydrogenase-2 reduces ischemic damage to the heart[J].Science, 2008, 321(5895):1493. doi: 10.1126/science.1158554
[17] GU JY, LI LW.ALDH2 Clu5O4Lys polymorphism and susceptibility to coronary artery disease and myocardial infarction in East Asians:a meta-analysis[J].Arch Med Res, 2014, 45(1):76. doi: 10.1016/j.arcmed.2013.10.002
[18] FANG T, CAO R, WANG W, et al.Alterations in necroptosis during ALDH2 mediated protection against high glucose induced H9c2 cardiac cell injury[J].Mol Med Rep, 2018, 18(3):2807.
[19] KANG PF, WU WJ, TANG Y, et al.Activation of ALDH2 with low concentration of ethanol attenuates myocardial ischemia/reperfusion injury in diabetes rat mode[J].Oxid Med Cell Longev, 2016, 2016:6190504.
[20] 胡龙江, 周音频, 曹运兰, 等.NLRP3炎性小体与冠心病的相关性[J].中国动脉硬化杂志, 2018, 26(3):281. doi: 10.3969/j.issn.1007-3949.2018.03.012
[21] 王秋悦.柔嫩艾美耳球虫rhomboid基因重组卡介苗的构建及其免疫保护性研究[D].长春: 吉林大学, 2009.