[1] 谢鹏.中枢神经系统感染[M]//贾建平.神经病学.6版.北京:人民卫生出版社，2010:233-236.
[2] 傅继华.病毒学实用实验技术[M].济南:山东科学技术出版社，2001:8082.
[3] 衣曼，魏克伦，王雪峰，等.小鼠单纯疽疹病毒性脑炎模型的建立[J].小儿急救医学，2000,7 (1):2627.
[4] 侯炜，杨占秋，肖红，等.单纯疽疹病毒脑炎模型的研究[J].动物医学进展，2003,24(2):106107.
[5] James SH, Kimberlin DW Whitley RJ. Antiviral therapy forherpesvirus central nervous system infecions: neonatal herpessimplex virus infection, herpes simplex encephalitis, and congenitalcytomegalovirus infection[J].Antiviral Res, 2009, 83 (3):207-213.
[6] Vilely MC, Campos RD, Mansur DS, et al. Role of 1L-4 in anexperimental model of encephalitis induced by intracranialinoculation of herpes simplex virus(HSV-1)[J].ArqNeuropsiquiatr,2011,69(2A):237241.
[7] Boivin G, Serqerie Y, Rivest S, et al. L,Effect of pretreatment withtoll-like receptor agonists in a mouse of herpes simplex virus type 1encephalitis[J].J lnfect Dis,2008,198 (5):664672.
[8] Armien AG, Hu S, Little MR, et al. Chronic cortical and subcorticalpathology with associated neurological deficits ensuing experimentalherpes encephalitis[J].Brain Patho1,2010,20 (4):738750.
[1] James G. Anesthetic pharmacology:reflections of a section editor[J].Anesth Analg,2007,105(5):11861190.
[2] Himmelseher S, Durieux ME Revising a dogma: ketamine forpatients with neurological injury?[J].Anesth Analg, 2005,101 (3):524534.
[3] Proescholdt M, Heimann A, Kempski O. Neuron protection of S(+)ketamine isomer in global forebrain ischemia[J].BrainRes,2001,904(3):245251.
[4] 吴优，贾建平.脑缺血动物模型的制各及影响因素[J].中国脑血管病杂志，2007,4 (1):2-44.
[5] 万丽，黄焕森，胡春旭，等.浅低温联合尼莫地平静脉输注对家兔全脑缺血再灌注损伤1L-1,TNF-, IL-10。表达的影响[J] . 临床麻醉学杂志，2009,25(6):507510.
[6] 施新酞.医学动物实验方法[M].北京:人民卫生出版社，1980:422.
[7] Joshi S, Wang M, Nishanian L,V. et al. Electrocerebral silence by intracarotid anesthetics does not affect early hyperemia after transient cerebral ischemia in rabbits[J].Anesth Analg, 2004, 98 (5):14541459.
[16] [8」张全云，石栩飒.利多卡因复合氯胺酮对全脑缺血大鼠海马 细胞凋亡的影响[J].国际麻醉学与复苏杂志，2009,30 (3) 206209.
[17] [9」何洹，施冲.静脉麻醉药物脑保护作用的研究进展[J].中国 微侵袭神经外科杂志，2008,13 (2):9496.
[10] 庄心良，曾因明，陈伯鉴.现代麻醉学[M] . 3版.北京:人民卫 生出版社，2009:2339.
[11] Monassier L, Tibirica E,Roegel JC, et al. Prevention by NMDA receptor antagonists of the centrally evoked increases of cardiac inotropic responses in rabbits [J].Br J Pharmacol, 1994, 111 (4):13471354.
[20] [12} Han J, Kim N, Joo H, et al. Ketamine abolishes ischemic preconditioning through inhibition of K (ATP) channels in rabbit hearts.AM J Physiol Heart Circ Physiol, 2002, 283(1): H13H21.
[13] Pietrzak B, Czarnecka E. Effect of combined administration of ethanol and tiagabine on rabbit L;L;G[J].Pharmacol Rep,2006, 58 (6):890899.
[14] Wang M, Agarwal S, Mayevsky A, et al. Optically measured NADH concentrations are unaffected by propofol induced L;L;G silence during transient cerebral hypoperfusion in anesthetized rabbits.Brain Res,2011，1396:6976.