[1] Lai L,C. Micro RNAs are complementary to 3' UTR sequencemotifs that mediate negative post-transcriptional regulation[J].Nat Genet,2002,30(4):363-364.
[2] Houbaviy hb, Murray Mf, Sharp PA. Embryonic stem cell specificmicroRNAs[J]. Dev Cell,2003,5(2):351-358.
[3] Lim LP, Lau NC, Weinstein EG, et al. The microRNAs ofcaenorhabditis elegans[J].Genes Dev,2003,17(8):991-1008.
[4] Barrel DP. MicroRNAs: genomies biogenesis, mechanism, andfunction[J].cell,2004,116(2):281-297.
[5] Calm GA, Sevignani C, Dumitru CD, et al. Human microRNAgenes are frequently located at fragile sites and genomic regionsinvolved in cancers[J].Proc Natl Acad Sci USA,2004,101 (9):2999-3004.
[6] Gaur A,,Jewell DA, Liang Y, et al. Characterization of microRNAexpression levels and their biological correlates in human cancercell lines[J].Cancer Res,2007,67 (6):2456-2468.
[7] Chan SH, Wu Cw, Li AF, et al. MiR-21 microRNA expression in humangastric carcinomas and its clinical association [J]. Anticancer Res, 2008, 28 (2A):907911.
[8]
[8] Zhang Z, Li Z, Gao C, et al. miR-2l plays a pivotal role in gastric cancer pathogenesis and progression.Lab lnvest, 2008, 88 (12):13581366.
[9] 吴炳坤，唐永德.荧光定量聚合酶链反应检测胃癌let-7 MicroRNA[J].检验医学，2008,23(3):305307.
[10] Johnson SM, Grosshans H, Shingara J, et al. RAS is regulated by the lets microRNA family[J].cell,2005,120(5):635647.
[11] Petrocca F, Visone R, Onelli MR, et al. E2F1-regulated microRNAs impair TCF beta-dependent cell-cycle arrest and apoptoms in gastric cancer[J].Cancer Cell, 2008，13 (3):272- 286.
[12] Katada T, lshiguro H, Kuwabara Y, et al. microRNA expression profile in undifferentiated gastric cancer[J].Int J Oncol, 2009, 34 (2) 537542.
[13] 林巧爱，李玲玲，巩文辞，等.miR-143在胃癌中的表达及其临 床病理意义[J].中国病理生理杂志，2010, 26 (9): 1674- 1678.
[14] Borralho PM, Kren BT, Castro RE, et al. MicroRNA-143 reduces viability and increase sensitivity to 5-fluorouracil in HTC116 human colorectal cancer cells[J].FEBS J, 2009, 276 (22): 66896700.
[l5] Lu,J, Getz G, Lric A, et al. MicroRNA expression profiles classify human cancer[J].Nature,2005,435(7043):834838.
[16] Li, Zhan W, Wang, et al. Inhibition of PRL-3 gene expression in gastric cacer cell line SGC7901 via microRNA suppressed reduces peritoneal metastasis[J].Biochem Biophys Res Commun, 2006, 348 (1):229237.
[17] Wang Z, He YL, Cai SR, et al. Expression and prognostic impact of PRL 3 in lymph node metastasis of gastric cancer: its molecular mechanism was investigated using artificial microRNA interference [J].Int J Cancer,2008,123 (6):14391447
[18] Motoyama K, moue H, Nakamura Y, et al. Clinical significance of high mobility group A2 in human gastric cancer and its relationship to let 7 microRNA family[J].Clin Cancer Res,2008, 14 (8):23342340.
[19] Kriitzfeldt j, Rniewsky N, Braich R, et al. Silencing of micro-RNAs in vivo with 'antagomirs'[J].Nature, 2005, 438 (7068):685- 689.
[20] Bandres L Bitarte N, Arias F, et al. microRNA-451 regulates macrophage migration inhibitory factor production and proliferation of gastrointestinal cancer cells.Clin Cancer Res, 2009, 15 (7):22812290.
[21] Xia L, Zhang D, Du R, et al. MiR-15b and miR-16 modulate multidrug resistance by targeting BCL2 in human gastric cancer cells[J].Int J Cancer,2008,123 (2):372379.
[22] Saito Y, Suzuki H, Hibi T. The role of microRNAs in gastro-intestinal cancers[J].J Gastroentero1,2009,44 (Suppl 19):1822