[1] ZHANG F, WEN Y, GUO X. CRISPR/Cas9 for genome editing: progress, implications and challenges[J]. Hum Mol Genet, 2014, 23(R1): R40.
[2] MA Y, ZHANG L, HUANG X. Genome modification by CRISPR/Cas9[J]. FEBS J, 2014, 281(23): 5186. doi: 10.1111/febs.13110
[3] GUPTA D, BHATTACHARJEE O, MANDAL D, et al. 2019, CRISPR-Cas9 system: a new-fangled dawn in gene editing[J]. Life Sci, 232: 116636. doi: 10.1016/j.lfs.2019.116636
[4] EYERS PA, KEESHAN K, KANNAN N. Tribbles in the 21st century: The evolving roles of tribbles pseudokinases in biology and disease[J]. Trends Cell Biol, 2017, 27(4): 284. doi: 10.1016/j.tcb.2016.11.002
[5] YOKOYAMA T, NAKAMURA T. Tribbles in disease: signaling pathways important for cellular function and neoplastic transformation[J]. Cancer Sci, 2011, 102(6): 1115. doi: 10.1111/j.1349-7006.2011.01914.x
[6] DU K, HERZIG S, KULKARNI RN, et al. TRB3: a tribbles homolog that inhibits Akt/PKB activation by insulin in liver[J]. Science, 2003, 300(5625): 1574. doi: 10.1126/science.1079817
[7] HUA F, HU ZW. TRIB3-P62 interaction, diabetes and autophagy[J]. Oncotarget, 2015, 6(33): 34061. doi: 10.18632/oncotarget.6108
[8] PRUDENTE S, BAILETTI D, MENDONCA C, et al. Infrequent TRIB3 coding variants and coronary artery disease in type 2 diabetes[J]. Atherosclerosis, 2015, 242(1): 334. doi: 10.1016/j.atherosclerosis.2015.07.030
[9] XU J, LV S, QIN Y, et al. TRB3 interacts with CtIP and is overexpressed in certain cancers[J]. Biochim Biophys Acta, 2007, 1770(2): 273. doi: 10.1016/j.bbagen.2006.09.025
[10] HUA F, SHANG S, YANG YW. TRIB3 interacts with β-Catenin and TCF4 to increase stem cell features of colorectal cancer stem cells and tumorigenesis[J]. Gastroenterology, 2019, 156(3): 708. doi: 10.1053/j.gastro.2018.10.031
[11] ZHANG X, ZHONG N, LI X, et al. TRIB3 promotes lung cancer progression by activating β-catenin signaling[J]. Eur J Pharmacol, 2019, 863: 172697. doi: 10.1016/j.ejphar.2019.172697
[12] HONG B, ZHOU J, MA K, et al. TRIB3 promotes the proliferation and invasion of renal cell carcinoma cells via activating MAPK signaling pathway[J]. Int J Biol Sci, 2019, 15(3): 587. doi: 10.7150/ijbs.29737
[13] QU J, LIU B, LI B, et al. TRIB3 suppresses proliferation and invasion and promotes apoptosis of endometrial cancer cells by regulating the AKT signaling pathway[J]. Onco Targets Ther, 2019, 12: 2235. doi: 10.2147/OTT.S189001
[14] ZHANG C, HONG FF, WANG CC, et al. TRIB3 inhibits proliferation and promotes osteogenesis in hBMSCs by regulating the ERK1/2 signaling pathway[J]. Sci Rep, 2017, 7(1): 10342. doi: 10.1038/s41598-017-10601-w
[15] 刘陶迪. 大鼠青春期前期精原干细胞自我更新和分化相关基因表达的初步研究[D]. 呼和浩特: 内蒙古大学, 2012.
[16] AIMÉ P, SUN X, ZAREEN N, et al. Trib3 is elevated in Parkinson′s Disease and mediates death in parkinson′s disease Models[J]. J Neurosci, 2015, 35(30): 10731. doi: 10.1523/JNEUROSCI.0614-15.2015
[17] ZHAO Y, HAN Y, BU DF, et al. Reduced AKT phosphorylation contributes to endoplasmic reticulum stress-mediated hippocampal neuronal apoptosis in rat recurrent febrile seizure[J]. Life Sci, 2016, 153: 153. doi: 10.1016/j.lfs.2016.04.008
[18] ZHANG J, HAN Y, ZHAO Y, et al. Inhibition of TRIB3 protects against neurotoxic injury induced by kainic acid in rats[J]. Front Pharmacol, 2019, 10: 585. doi: 10.3389/fphar.2019.00585
[19] SOWERS JR. Role of TRIB3 in diabetic and overnutrition-induced atherosclerosis[J]. Diabetes, 2012, 61(2): 265. doi: 10.2337/db11-1495
[20] WANG S, WANG C, LI X, et al. Down-regulation of TRIB3 inhibits the progression of ovarian cancer via MEK/ERK signaling pathway[J]. Cancer Cell Int, 2020, 20: 418. doi: 10.1186/s12935-020-01509-z